It is important to note that the entrance of amniotic fluid and fetal material into the bloodstream is a normal part of the birth process and in most women, it does not cause this serious reaction. It is believed to occur when a mother suffers an allergic-like (immune) response to amniotic fluid and fetal material that enters her bloodstream during labor or shortly after delivery. His one great achievement is being the father of three amazing children.Although the exact mechanism of how an amniotic fluid embolism (AFE) occurs is not well understood. He is one of the founders of the FOAM movement (Free Open-Access Medical education) and is co-creator of , the RAGE podcast, the Resuscitology course, and the SMACC conference. He created the ‘Critically Ill Airway’ course and teaches on numerous courses around the world. He coordinates the Alfred ICU’s education and simulation programmes and runs the unit’s education website, INTENSIVE. He is actively involved in in using translational simulation to improve patient care and the design of processes and systems at Alfred Health.
Dic amniotic fluid embolism professional#
He has completed fellowship training in both intensive care medicine and emergency medicine, as well as post-graduate training in biochemistry, clinical toxicology, clinical epidemiology, and health professional education. He is an internationally recognised Clinician Educator with a passion for helping clinicians learn and for improving the clinical performance of individuals and collectives.Īfter finishing his medical degree at the University of Auckland, he continued post-graduate training in New Zealand as well as Australia’s Northern Territory, Perth and Melbourne. He is on the Board of Directors for the Intensive Care Foundation and is a First Part Examiner for the College of Intensive Care Medicine. He is also a Clinical Adjunct Associate Professor at Monash University. He is a co-founder of the Australia and New Zealand Clinician Educator Network (ANZCEN) and is the Lead for the ANZCEN Clinician Educator Incubator programme. Oxytocics + bimanual massage & uterine packing +/- hysterectomy may be indicated.Ĭhris is an Intensivist and ECMO specialist at the Alfred ICU in Melbourne.Early delivery of fetus vital for both patients – if mum having CPR delivery within 5 min.C – CPR + left uterine displacement, IVF, inotropes, PAC may be needed -> initially develop right heart failure then this recovers and left heart begins to fail -> treat left sided heart failure aggressively.Call for help – anaesthetic, obstetric, haematological, ICU, paediatric, midwifery.plasma concentration of zinc coproporphyrin (a component of meconium).specific antigen testing of maternal bloods can help.
No typical presentation! -> ‘Classic’ triad = hypoxia, haemodynamic collapse, DIC within 30min -> complement activation + intense pulmonary vasoconstriction -> right heart failure -> hypoxia, hypercarbia & acidosis -> left heart failure + APO.intravascular entry of prostaglandins, leukotrienes, endothelin and fetal debris.probably due to an anaphylactic reaction to fluid or fetal tissue.mortality 50% in first hour -> if patients survive usually have neurological deficits.
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